Full Length Research Paper
References
Prasad AJM, Kemparaju K, Elizabeth A. Frank and Cletus J. M. D'Souza (2009). Purification of human serum paraoxonase: A simple and rapid method. Afr. J. Biochem. Res. 3(4):125-129. | ||||
Anker SD, Egerer KR, Volk HD, Kox WJ, Poole-Wilson PA, Coats AJ (1997). Elevated soluble CD14 receptors and altered cytokines in chronic heart failure. Am. J. Cardiol. 79:1426-1430. Crossref |
||||
Aviram M, Maor I, Keidar S (1995). Lesioned low density lipoprotein in atherosclerotic apolipoprotein E-deficient transgenic mice and in humans is oxidized and aggregated. Biochem. Biophys. Res. Commun. 216:501-513. Crossref |
||||
Aviram M, Rosenblat M (2008). Paraoxonases (PON1, PON2, PON3) analyses in vitro and in vivo in relation to cardiovascular diseases. Methods Mol. Biol. 477:259-76. Crossref |
||||
Aviram M, Rosenblat M, Bisgaier CL, Newton RS, Primo-Parmo SL, La Du BN (1998). Paraoxonase inhibits high-density lipoprotein oxidation and preserves its functions: a possible peroxidative role for paraoxonase. J. Clin. Invest. 101:1581-1590. Crossref |
||||
Aviram M, Rosenblat M, Billecke S, Erogul J, Sorenson R, Bisgaier CL, Newton RS, La Du B (1999). Human serum paraoxonase (PON 1) is inactivated by oxidized low density lipoprotein and preserved by antioxidants. Free Radic. Biol. Med. 26(7-8):892-904. Crossref |
||||
Aviram M, Hardak E, Vaya J, Mahmood S, Milo S, Hoffman A, Billicke S, Draganov D, Rosenblat M (2000). Human serum paraoxonase (PON1) Q and R selectively decrease lipid peroxides in human coronary and carotid atherosclerotic lesions. Circulation 101:2510-2517. Crossref |
||||
Azarsiz E, Kayikcioglu M, Payzin S, Sözmen EY (2003). PON1 activities and oxidative markers of LDL in patients with angiographically proven coronary artery disease. Int. J. Cardiol. 91:43-51. Crossref |
||||
Bonagura JD (1983). M-Mode echocardiography: basic principles. Vet. Clin. North Am. Small Anim. Pract. 13(2):299-319. Pubmed |
||||
Burton KP, McCord JM, Ghai G (1984). Myocardial alterations due to free-radical generation. Am. J. Physiol. 246:776-783. |
||||
Bionaz M, Trevisi E, Calamari L, Librandi F, Ferrari A, Bertoni G (2007). Plasma paraoxonase, health, inflammatory conditions and liver function in transition dairy cows. J. Dairy Sci. 90(4):170-176. Crossref |
||||
Canales A, Sanchez-Muniz FJ (2003). Paraoxonase, something more than an enzyme? Med. Clin. 121(14):537-548. Crossref |
||||
Chetboul VT, Nicolle A, sampederano CC, Gouni V, pouchelon JL, Lefebvre HP, Concordet D (2005). Effects of animal position and number of repeated measurements on selected two-dimensional and M-mode echocardiographic variables in healthy dogs." J. Am. Vet. Med. Assoc. 227(5):743-747. Crossref |
||||
Dhalla AK, Hill MF, Singal PK (1996). Role of oxidative stress in transition of hypertrophy to heart failure. J. Am. Coll. Cardiol. 28:506-514. Crossref |
||||
Feingold K, Memon RA, Moser AH, Grunfeld C (1998). Paraoxonase activity in the serum and hepatic mRNA levels decrease during the acute phase response. Atherosclerosis 139(2):307-315. Crossref |
||||
Förstermann U (2010). Nitric oxide and oxidative stress in vascular disease. Pflugers Arch. 459(6):923-939. Crossref |
||||
Fox P, Moise NS (1998). Primary Myocardial disease in dogs. Text Book of Canine and feline cardiology. pp. 582-617. | ||||
Fox P (1989). Myocardial disease textbook of Veterinary internal medicine. P 1097. | ||||
Gan KN, Smolen A, Eckerson HW (1991). Purification of human serum paraoxonase/arylesterase: evidence for one esterase catalyzing both activities. Drug Metab. Dispos. 19:100-106. PMid:1673382 |
||||
Genest J Jr, McNamara JR, Salem DN, Ordovas JM, Jenner JL, Millar JS, Silberman SR, Wilson PF, Schaefer EJ (1992). Lipoprotein cholesterol, apolipoproteins A-I and B, and Lipoprotein (a) in men with premature coronary artery disease. J. Am. Coll. Cardiol. 19:782-802. Crossref |
||||
Goldhaber JI, Ji S, Lamp ST (1989). Effects of exogenous free radicals on electromechanical function and metabolism in isolated rabbit and guinea pig ventricle. J. Clin. Invest. 83:1800-1809. Crossref |
||||
Granér M, James RW, Kahri J, Nieminen MS, Syvänne M, Taskinen MR (2006). Association of paraoxonase-1 activity and concentration with angiographic severity and extent of coronary artery disease. J. Am. Coll. Cardiol. 47:2429-2435. Crossref |
||||
Griendling KK, Sorescu D, Ushio-Fukai M (2000). NAD(P)H oxidase: role in cardiovascular biology and disease. Circ. Res. 86:494-501. Crossref |
||||
Guerra L, Cerbai E, Gessi S, Borea PA, Mugello A (1996). The effect of oxygen free radicals on calcium current and dihydropyridine binding sites in guinea-pig ventricular myocytes. Br. J. Pharmacol. 118:1278-1284. Crossref |
||||
Hassett C, Richter RJ, Humbert R, Chapline C, Crabb JW, Omiecinski CJ, Furlong CE(1991). Characterization of cDNA clones encoding rabbit and human serum paraoxonase: The mature protein retains its signal sequence. Biochemistry 30:10141-9. Crossref |
||||
Hill MF, Singal PK (1997). Right and left myocardial antioxidant responses during heart failure subsequent to myocardial infarction. Circulation 96:2414-2420. Crossref |
||||
Jarvik GP, Tsai NT, McKinstry LA, Wani R, Brophy VH, Richter RJ, Schellenberg GD, Heagerty PJ, Hatsukami TS, Furlong CE (2002). Vitamin C and E Intake is associated with increasesed paraoxoanase activity. Arterioscler Thromb. Vasc. Biol. 22(8):1329-33. Crossref |
||||
Katsuramaki T, Hirata K, Kimura Y, Nagayama M, Meguro M, Kimura H, Honma T, Furuhata T, Hideki U, Hata F, Mukaiya M (2002). Changes in serum levels of apolipoprotein A-1 as an indicator of protein metabolism after hepatectomy. Wound Repair Regen. 10:77-8. Crossref |
||||
Keith M, Geranmayegan A, Sole MJ (1998). Increased oxidative stress in patients with congestive heart failure. J. Am. Coll. Cardiol. 31:1352-1356. Crossref |
||||
Kittleson MK (1998). Primary myocardial disease. Small animal cardiovascular medicine. Elsevier Health. | ||||
Mackness MI, Durrington PN, Ayub A, Mackness B (1999). Low serum paraoxonase: a risk factor for atherosclerotic disease? Chem. Biol. Interact 119(120):389-397. Crossref |
||||
Mackness B, Davies GK, Turkie W, Lee E, Roberts DH, Hill E (2001) Paraoxonase status in coronary heart disease. Are activity and concentration more important than genotype? Atrerioscler. Thromb. Vasc. Biol. 21:1451-1457. Crossref |
||||
Mak S, Newton GE (2001). The Oxidative Stress Hypothesis of Congestive Heart Failure. Chest 120:2035-2046. Crossref |
||||
Mallat Z, Philip I, Lebret M (1998). Elevated levels of 8-iso-prostaglandin F2α in pericardial fluid of patients with heart failure: a potential role for in vivo oxidant stress in ventricular dilatation and progression to heart failure. Circulation 97:1536-1539. Crossref |
||||
Monnet EO, Salman M, Boon J (1995). Idiopathic dilated cardiomyopathy in dogs: Survival and prognostic indicators. J. Vet. Med. 9(1):12-17. |
||||
The Criteria Committee of the New York Heart Association. Nomenclature and Criteria for Diagnosis of Diseases of the Heart and Great Vessels. 9th ed. Boston, Mass: Little, Brown & Co; 253-256 | ||||
Oda MN, Bielicki TT, Ho T, Berger EM, Rubin T, Forte M (2001). Paraoxonase 1 overexpression in mice and its effect on high-density lipoproteins. Biochem. Biophys. Res. Commun. 290:921-927. Crossref |
||||
Schrier GM, Hess ML (1988). Quantitative identification of superoxide anion as a negative inotropic species. Am. J. Physiol. 255:138-143. |
||||
Sisson DD, Thomas WP and Keene BW (2000) Primary Myocardial disease in the dogs. In text book of veterinary internal medicine. S.J.E. E.C Feldam. Philadelphia. WB Saunders Co. pp. 874-895. | ||||
Sisson DT, WP (1995). Myocardial diseases. Textbook of Veterinary internal medicine. WB Saunders Co. pp. 995-1005. | ||||
Thomas WP (1984). Two dimensional, real-time echocardiography in the dog: technique and validation. Vet. Radiol. 2:50-64. Crossref |
||||
Thomas WG, Jacobs GJ, Kaplan PM, Lombard CW, Moise NS, Moses BL (1993). Recommendations for standards in transthoracic two-dimensional echocardiography in the dog and cat. Echocardiography Committee of the Specialty of Cardiology American College of Veterinary Internal Medicine." J. Vet. Int. Med. 7(4):247-252. Crossref |
||||
Turk R, Juretic D, Geres D, Turk N, Rekic B, Simeon-Rudolf V, Svetina A (2004). Serum Paraoxonase activity and lipid parameters in early postpartum period of dairy cows. Res. Vet. Sci. 76:57-61. Crossref |
||||
Turk RD, Juretic D, Geres N, Turk B, Rekic V, Simeon-Rudolf M, Rebic B, Svetina A (2005). Serum Paraoxonase activity in dairy cows during pregnancy. Res. Vet. Sci. 79:15-18. Crossref |
||||
Turk RD, Juretic D, Geres A, Svetina N, Turk N, Flegar-Mestric Z (2007). Influence of Oxidative stress and metabolic adaptation on PON1 activity and MDA levels in Transition dairy cows . Anim. Reprod. Sci. 108(1-2):98-106. Crossref |
||||
Tsutsui H, Kinugawa S, Matsushima S (2008). Oxidative stress and mitochondrial DNA damage in heart failure. Cir. J. 72:31-37. Crossref |
||||
Watson AD, Berliner JA, Hama SY (1995). Protective effect of high density lipoprotein associated paraoxonase: inhibition of the biological activity of minimally oxidized low-density lipoprotein. J. Clin. Invest. 96:2882-2891. Crossref |
||||
Xie XM, Zhao SP (2002). Congestive heart failure and paraoxonase. 27(2):157-158. |
Copyright © 2022 Author(s) retain the copyright of this article.
This article is published under the terms of the Creative Commons Attribution License 4.0