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References
Adcocks C, Collin P, Buttle DJ (2002). Catechins from green tea (Camellia sinensis) inhibit bovine and human cartilage proteoglycan and type II collagen degradation in vitro. J. Nutr. 132:341-346. Pubmed |
||||
Ahmed S, Marotte H, Kwan K, Ruth JH, Campbell PL, Rabquer BJ, Pakozdi A, Koch AE (2008). Epigallocatechin-3-gallate inhibits IL-6 synthesis and suppresses trans signaling by enhancing soluble gp130 production. Proc. Natl. Acad. Sci. USA105:14692–7. Crossref |
||||
Ahmed S, Rahman A, Hasnain A, Lalonde M, Goldberg VM, Haqqi TM (2002). Green tea polyphenol epigallocatechin-3-gallate inhibits the IL-1β-induced activity and expression of cyclooxygenase-2 and nitric oxide synthase-2 in human chondrocytes. Free Radic. Biol. Med. 33:1097-1105. Crossref |
||||
Ahmed S, Wang N, Lalonde M, Goldberg VM, Haqqi TM (2004). Green tea polyphenol epigallocatechin-3-gallate (EGCG) differentially inhibits interleukin-1 beta-induced expression of matrix metalloproteinase-1 and-13 in human chondrocytes. J. Pharmacol. Exp. Ther. 308:767-773. Crossref |
||||
Alschuler L (1998). Green Tea: Healing tonic. Am. J. Nat. Med. 5:28-31. | ||||
Arab L, Il'yasova D (2003).The epidemiology of tea consumption and colorectal cancer incidence. J. Nutr. 133:S3310–8. |
||||
Arnett FC, Edworthy SM, Bloch DA, McShane DJ, Fries JF,Cooper NS, Healey LA, Kaplan SR, Liang MH, Luthra HS, Medsger TA Jr., Mitchell DM, Neustadt DH, Pinals RS, Schaller JG, Sharp JT, Wilder RL, Hunder GG (1988). The American Rheumatism Association 1987 revised criteria for the classification of rheumatoid arthritis. Arthritis Rheum. 31:315–324. Crossref |
||||
Berman B (2004). Complementary and alternative medicine: is it just a case of more tools for the medical bag? Clin. J. Pain 20:1–2. Crossref |
||||
Brosseau L, Wells GA, Tugwell P, Egan M, Wilson KG, Dubouloz CJ, Casimiro L, Robinson VA, McGowan J, Busch A, Poitras S, Moldofsky H, Harth M, Finestone HM, Nielson W, Haines-Wangda A, Russell-Doreleyers M, Lambert K, Marshall AD, Veilleux L; Ottawa Panel Members (2004). Ottawa panel evidence-based clinical practice guidelines for therapeutic exercises in the management of rheumatoid arthritis in adults. Phys. Ther. 84(10):934–972. | ||||
Chen JH, Tipoe GL, Liong EC, So HS, Leung KM, Tom WM, Fung PC, Nanji AA (2004). Green tea polyphenols prevent toxin-induced hepatotoxicity in mice by down-regulating inducible nitric oxide–derived prooxidants. Am. J. Clin. Nutr. 80:742-751. Pubmed |
||||
Choy EH, Panayi GS (2001). Cytokine pathways and joint infl ammation in rheumatoid arthritis. N. Engl. J. Med. 344: 907-16. Crossref |
||||
Cooper R, Morre DJ, Morre DM (2005). Medicinal benefits of green tea: part I. Review of non-cancer health benefits. J. Altern. Compl. Med. 11:521-528. Crossref |
||||
den Broeder AA, Joosten L, Saxne T, Heinegard D, Fenner H, Miltenburg A, Frasa W, van Tits LJ, Buurman W, van Riel PLCM, van de Putte LBA, Barrera P (2002). Long term anti-tumour necrosis factor alpha monotherapy in rheumatoid arthritis: effect on radiological course and prognostic value of markers of cartilage turnover and endothelial activation. Ann Rheum Dis.61:311-8. Crossref |
||||
Dessein PH, Norton GR, Woodiwiss AJ, Joffe BI, Solomon A (2007). Independent role of conventional cardiovascular risk factors as predictors of C-reactive protein concentrations in rheumatoid arthritis. J. Rheumatol. 34:681–8. Pubmed |
||||
Di Cesare PE, Fang C, Leslie MP, Della Valle CJ, Gold JM, Tulli H, Perris R, Carlson CS (1999). Localization and expression of cartilage oligomeric matrix protein by human rheumatoid and osteoarthritic synovium and cartilage. J. Orthop. Res. 17:437-445. Crossref |
||||
Dingle JT (1991). Cartilage maintenance in osteoarthritis: interaction of cytokines, NSAID and prostaglandins in articular cartilage damage and repair. J. Rheumatol. 18: 30–37. |
||||
Doss MX, Potta SP, Hescheler J, Sachinidis A (2005). Trapping of growth factors by catechins: a possible therapeutical target for prevention of proliferative diseases. J. Nutr. Biochem. 16:259-266. Crossref |
||||
Dube B, Rita T B, Satya P, Kalra A, Michael G (2008). Increased leptin expression selectively in the hypothalamus suppresses inflammatory markers CRP and IL-6 in leptin-deficient diabetic obese mice. Peptides 29: 593-598. Crossref |
||||
Dulloo AG, Seydoux J, Girardier L, Chantre P, Vandermander J (2000). Green tea and thermogenesis: interactions between catechin polyphenols, caffeine, and sympathetic activity. Int. J. Obes. Relat. Metab. Disord. 24:252-258. Crossref |
||||
Ekdahl C, Broman G (1992). Muscle strength, endurance, and aerobic capacity in rheumatoid arthritis: a comparative study with healthy subjects. Ann. Rheum. Dis. 51(1):35–40. Crossref |
||||
Erba D, Riso P, Bordoni A, Foti P, Biagi PL, Testolin G (2005). Effectiveness of moderate green tea consumption on antioxidative status and plasma lipid profile in humans. J. Nutr. Biochem. 16:144 –9. Crossref |
||||
Feldmann M, Maini SR (2008).Role of cytokines in rheumatoid arthritis: an education in pathophysiology and therapeutics. Immunol. Rev. 223:7-19. Crossref |
||||
Firesten GS (2004). The T cell cometh: Interplay between adaptive immunity and cytokine networks in rheumatoid arthritis. J. Clin. Invest. 114: 471-4. Crossref |
||||
Fransen J, Stucki G, Van Rielplc M (2003). Rheumatoid arthritis measures. Arthritis Rheum. 49 (Suppl.5):S214-24. Crossref |
||||
Frei B, Higdon JV (2003). Antioxidant activity of tea polyphenols in vivo: evidence from animal studies. J. Nutr. 133:3275S-84S. Pubmed |
||||
Graham HN (1992). Green tea composition, consumption, and polyphenol chemistry. Prev. Med. 21:334-350. Crossref |
||||
Haqqi TM, Anthony DD, Gupta S, Ahmad N, Lee MS, Kumar GK, Mukhtar H (1999). Prevention of collagen-induced arthritis in mice by a polyphenolic fraction from green tea. Proc. Natl. Acad. Sci. USA 96:4524-4529. Crossref |
||||
Hitchon CA, Alex P, Erdile LB, Frank MB, Dozmorov I, Tang Y, Wong K, Centola M, El-Gabalawy HS (2004). A distinct multi-cytokine profi le is associated with anti-cyclical citrullinated peptide antibodies in patients with early untreated inflammatory arthritis. J. Rheumatol. 31:2336-46. Pubmed |
||||
Hoff JF, Singlengleton KIA (1997). A method for determination of tannin in foods by means of immobilized enzymes. J. Food Sci. 42:1566. Crossref |
||||
Hye SP, Jung YP, Rina Y (2005). Relationship of obesity and visceral adiposity with serum concentrations of CRP, TNF-α and IL-6. Diabetes Res. Clin. Pract. 69:29-35. Crossref |
||||
Inoue E, Yamanaka H, Hara M, Tomatsu T, Kamatani N (2007). Comparison of Disease Activity Score (DAS)-28- erythrocyte sedimentation rate and DAS28- C-reactive protein threshold values. Ann. Rheum. Dis. 66:407–409. Crossref |
||||
Juhel C, Armand M, Pafumi Y, Rosier C, Vandermander J, Lairon D (2000). Green tea extract (AR25) inhibits lipolysis of triglycerides in gastric and duodenal medium in vitro. J. Nutr. Biochem. 11:45-51. Crossref |
||||
Khokhar S, Magnusdottir SGM (2002). Total phenol, catechin and caffeine contents of teas commonly consumed in the United Kingdom. J. Agric. Food Chem. 50:565. Crossref |
||||
Kim S, Lee MJ, Hong J, Li C, Smith TJ, Yang GY, Seril DN, Yang CS (2000). Plasma and tissue levels of tea catechins in rats and mice during chronic consumption of green tea polyphenols. Nutr. Cancer 37:41-48. Crossre |
||||
Kremers HM, Nicola P, Crowson CS, Michael O' fallon W, Gabriel SE (2004). Therapeutic strategies in rheumatoid arthritis over a 40-year period. J. Rheumatol. 31:2366–2373. Pubmed |
||||
Kroot EJ1, de Jong BA, van Leeuwen MA, Swinkels H, van den Hoogen FH, van't Hof M, van de Putte LB, van Rijswijk MH, van Venrooij WJ, van Riel PL (2000). The prognostic value of anti-cyclic citrullinated peptide antibody in patients with recent-onset rheumatoid arthritis. Arthritis Rheum. 43:1831-1835. Crossref |
||||
Lambert JD, Sang S, Yang CS (2007). Biotransformation of green tea polyphenols and the biological activities of those metabolites. Mol. Pharm. 4:819–25. Crossref |
||||
Lin RW, Chen CH, Wang YH, Ho ML, Hung SH, Chen IS, Wang GJ (2009). (–)-Epigallocatechin gallate inhibition of osteoclastic diff erentiation via NF-κB. Biochem. Biophys. Res. Commun. 379:1033-1037. Crossref |
||||
Lin SK, Chang HH, Chen YJ, Wang CC, Galson DL, Hong CY, Kok SH (2008). Epigallocatechin-3-gallate diminishes CCL2 expression in human osteoblastic cells via up-regulation of phosphatidylinositol 3-Kinase/Akt/ Raf-1 interaction: a potential therapeutic benefit for arthritis. Arthritis Rheum. 58:3145-3156. Crossref |
||||
Lipsky PE (2005). Rheumatoid arthritis. In: Kasper D, Braunwald E, Fauci A, Hauser S, Longo D, Jameson J (eds.), Harrison's principles of internal medicine, 16th ed. New York: McGraw-Hill. | ||||
Majeed M, McQueen F, Yeoman S, McLean L (2004).Relationship between serum hyaluronic acid level and disease activity in early rheumatoid arthritis. Ann. Rheum. Dis. 63:1166-1168 doi:10.1136/ard.2003.010942 Crossref |
||||
Maki KC, Reeves MS, Farmer M, Yasunaga K, Matsuo N, Katsuragi Y, Komikado M, Tokimitsu I, Wilder D, Jones F, Blumberg JB, Cartwright Y (2009). Green tea catechin consumption enhances exercise-induced abdominal fat loss in overweight and obese adults. J. Nutr. 139:264-270. Crossref |
||||
Malemud CJ (2004). Cytokines as therapeutic targets for osteoarthritis. Biol. Drugs 18:23-35. Crossref |
||||
Malemud CJ (2006). Matrix metalloproteinases: role in skeletal development and growth plate disorders. Front. Biosci. 11:1702-15. Crossref |
||||
Manning J, Roberts JC (2003). Analysis of catechin content of commercial green tea products. J. Herbal Pharmacother. 3:19-32. Crossref |
||||
Månsson B, Carey D, Alini M, Ionescu M, Rosenberg LC, Poole AR, Heinegård D, Saxne T (1995).Cartilage and bone metabolism in rheumatoid arthritis. Differences between rapid and slow progression of disease identified by serum markers of cartilage metabolism. J. Clin. Invest. 95:1071-7. Crossref |
||||
McInnes IB (2001). Rheumatoid arthritis: From bench to bedside. Rheum. Dis. Clinics North Am. 27(2):373–387. Crossref |
||||
McKenna DJ, Hughes K, Jones K (2000). Green tea monograph. Altern. Ther. Health Med. 6:61–8, 70–2, 4. |
||||
Morinobu A, Biao W, Tanaka S, Horiuchi M, Jun L, Tsuji G, Sakai Y, Kurosaka M, Kumagai S (2008). (–)-Epigallocatechin-3-gallate suppresses osteoclast differentiation and ameliorates experimental arthritis in mice. Arthritis Rheum. 58:2012-2018. Crossref |
||||
Morozzi G, Fabbroni M, Bellisai F, Cucini S, Simpatico A, Galeazzi M (2007). Low serum level of COMP, a cartilage turnover marker, predicts rapid and high ACR70 response to adalimumab therapy in rheumatoid arthritis. Clin. Rheumatol. 26:1335–1338. Crossref |
||||
Mundermann A, Dyrby CO, Andriacchi TP, King KB (2005). Serum concentration of cartilage oligomeric matrix protein (COMP) is sensitive to physiological cyclic loading in healthy adults. Osteoarthritis Cartilage 13:34–38. Crossref |
||||
Natsume H, Adachi S, Takai S, Tokuda H, Matsushima-Nishiwaki R, Minamitani C, Yamauchi J, Kato K, Mizutani J, Kozawa O, Otsuka T (2009). (–)-Epigallocatechin gallate attenuates the induction of HSP27 stimulated by sphingosine 1-phosphate via suppression of phosphatidylinositol 3-kinase/Akt pathway in osteoblasts. Int. J. Mol. Med. 24:197-203. Pubmed |
||||
Niki Y, Takeuchi T, Nakayama M, Nagasawa H, Kurasawa T, Yamada H, Toyama Y, Miyamoto T (2012). Clinical Significance of Cartilage Biomarkers for Monitoring Structural Joint Damage in Rheumatoid Arthritis Patients Treated with Anti-TNF Therapy. PLoS ONE 7(5):e37447. Crossref |
Nutrition and Your Health: Dietary Guidelines for Americans (1995). U.S. Department of Agriculture, U.S. Department of Health and Human Services. 4th edition. | ||||
Ohinishi M, Morishita H, Iwahashi H, Shizuo T, Yoshiaki S, Kimura M, Kido R (1994). Inhibitory effects of chologenic acids on linoleic acid peroxidation and haemolysis. Phytochemistry 36:579-583. Crossref |
||||
Patrick B, Tobias B, Ru¨diger H, Georg B, Roland J, Felix B, Michael S, Heike N (2010). Dietinduced obesity, exogenous leptin-, and MADB106 tumor cell challenge affect tissue leukocyte distribution and serum levels of cytokines in F344 rats. Endocrinology 38:104–112. | ||||
Pope RM (2002). Apoptosis as a therapeutic tool in rheumatoid arthritis. Nat. Rev. Immunol. 2:527-535. Crossref |
||||
Ramesh E, Geraldine P, Thomas PA (2009). Regulatory effect of epigallocatchin gallate on the expression of C-reactive protein and other inflammatory markers in an experimental model of atherosclerosis. Chem. Biol. Interact. [Epub ahead of print]. | ||||
Ravishankara M N, Shrivastava N, Padh H, Rajani M (2002). Evaluation of antioxidant properties of root bark of Hemidesmus indicus R. Br. (Anantmul). Phytomedicine 9:153-160. Crossref |
||||
Riegsecker S, Wiczynski D, Kaplan MJ, Ahmed S (2013). Potential benefits of green tea polyphenol EGCG in the prevention and treatment of vascular inflammation in rheumatoid arthritis. Life Sci. 3;93(8):307-12. |
||||
Sabu MC, Priya TT, Ramadsan K, Ikuo Nishigaki1 I (2010). Beneficial effect of green tea; A literature review. Chin. Med 5:13. Crossref |
||||
Sattar N, McCarey DW, Capell H, McInnes IB (2003). Explaining how "high-grade "systemic inflammation accelerates vascular risk in rheumatoid arthritis. Circulation 108(24):2957–2963. Crossref |
||||
Saxne T, Månsson B (2000). Molecular markers for assessment of cartilage damage in rheumatoid arthritis. In: Firestein G, Panayi GS, Wollheim FA (eds.), Rheumatoid Arthritis: New Frontiers in Pathogenesis and Treatment. Oxford: Oxford University Press. pp. 291-304. | ||||
Singh R, Ahmed S, Islam N, Victor M. Goldberg VM, Haqqi TM (2002). Epigallocatechin-3- gallate inhibits interleukin-1β-induced expression of nitric oxide synthase and production of nitric oxide in human chondrocytes: suppression of nuclear factor κB activation by degradation of the inhibitor of nuclear factor κB. Arthritis Rheum. 46:2079-2086. Crossref |
||||
Skoumal M, Haberhauer G, Feyertag J, Kittl EM, Bauer K, Dunky A (2004). Serum levels of cartilage oligomeric matrix protein are elevated in rheumatoid arthritis, but not in inflammatory rheumatic diseases such as psoriatic arthritis, reactive arthritis, Raynaud's syndrome, scleroderma, systemic lupus erythematosus, vasculitis and Sjögren's syndrome. Arthritis Res. Ther. 6:73-74. Crossref |
||||
Skoumal M, Kolarz G, Klingler A (2003). Serum levels of cartilage oligomeric matrix protein. A predicting factor and a valuable parameter for disease management in rheumatoid arthritis. Scand. J. Rheumatol. 32:156-61. Crossref |
||||
Smith JB, Haynes MK (2002). Rheumatoid arthritis; a molecular understanding. Ann. Intern. Med. 136:908-22. Crossref |
||||
Smolen JS, Breedveld FC, Schiff MH, Kalden JR, Emery P, Eberl, G, van Riel PL, Tugwell P (2003). A simplified disease activity index for rheumatoid arthritis for use in clinical practice. Rheumatology 42:244–57. Crossref |
||||
Sokka T, Häkkinen A, Kautiainen H, Maillefert JF, Toloza S, Mørk Hansen T, Calvo-Alen J, Oding R, Liveborn M, Huisman M, Alten R, Pohl C, Cutolo M, Immonen K, Woolf A, Murphy E, Sheehy C, Quirke E, Celik S, Yazici Y, Tlustochowicz W, Kapolka D, Skakic V, Rojkovich B, Müller R, Stropuviene S, Andersone D, Drosos AA, Lazovskis J, Pincus T; QUEST-RA Group (2008). Physical inactivity in patients with rheumatoid arthritis: data from twenty-one countries in a cross-sectional, international study. Arthritis Rheum. 59:42–50. Crossref |
||||
Takai S, Matsushima-N R, Adachi S, Natsume IH, Minamitani C, Mizutani J, Otsuka T, Tokuda H, Kozawa O (2008). (–)-Epigallocatechin gallate reduces platelet-derived growth factor-BB-stimulated interleukin-6 synthesis in osteoblasts: suppression of SAPK/JNK. Mediators Inflamm. 2008: 291808. Crossref |
||||
Takeuchi T, Yamanaka H, Inoue E, Nagasawa H, Nawata M, Ikari K, Saito K, Sekiguchi N, Sato E, Kameda H, Iwata S, Mochizuki T, Amano K, Tanaka Y (2008). Retrospective clinical study on the notable efficacy and related factors of infliximab therapy in a rheumatoid arthritis management group in Japan: one year outcome of joint destruction (RECONFIRM-2J). Mod Rheumatol. 18:447–454. Crossref |
||||
Vankemmelbeke MN, Jones GC, Fowles C, Ilic MZ, Handley CJ, Day AJ, Knight CG, Mort JS, Buttle DJ (2003). Selective inhibition of ADAMTS-1, -4 and -5 by catechin gallate esters. Eur. J. Biochem. 270:2394-2403. Crossref |
||||
Wahyudi S, Sargowo D (2007). Green tea polyphenols inhibits oxidized LDL- induced NKKB activation in human umbilical vein endothelial cell. Acta Med. Indones. 39(2): 66-70. Pubmed |
||||
Waskmundzka M, Wianowska D, Szewczyk K, Oniszczuk A (2007). Effect of sample-preparation methods on the HPLC quantitation of some phenolic acids in plant materials. Acta Chromatographica 19:227-237. | ||||
Yang MH, Wang CH, Chen HL (2001). Green tea, Oolong and black tea extract modulate lipid metabolism in hyper lipidemic rats fed high sucrose diet. J. Nutr. Biochem. 12:14-20. Crossref |
||||
Yen GC, Chen HY (1997). Antioxidant activity of various tea extracts in relation to their mutagenicity. J. Agric. Food Chem. 43:27. Crossref |
||||
Yokozawa T, Dong E, Nakagawa T, Kashiwagi H, Nakagawa H, Takeuchi S, Chung HY (1998). In vitro and in vivo studies on the radical-scavenging activity of tea. J. Agric. Food Chem. 46:2143. Crossref |
||||
Young-Min S, Cawston T, Marshall N, CoadyD, Christgau S, Saxne T, Robins S, Griffiths I (2007). Biomarkers predict radiographic progression in early rheumatoid arthritis and perform well compared with traditional markers. Arthritis Rheum. 56:3236–3247. Crossref |
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